When renin is released from the juxtaglomerular cells
O blood flow to the kidneys decreases.
O angiotensinogen is converted into angiotensin I.
O angiotensin I is converted to angiotensin II.
O angiotensin I increases salt reabsorption at the kidneys.
O blood pressure goes down.
O angiotensinogen is converted into angiotensin I.
When renin is released from the juxtaglomerular cells, several physiological responses occur:
1. Angiotensinogen is converted into angiotensin I: Renin acts on angiotensinogen, a protein produced by the liver, and converts it into angiotensin I.
2. Angiotensin I is converted to angiotensin II: Angiotensin-converting enzyme (ACE) present in the lungs and other tissues converts angiotensin I into angiotensin II. This conversion is critical for the activation of angiotensin II.
3. Angiotensin II has various effects: Angiotensin II is a potent vasoconstrictor, causing narrowing of the blood vessels. This vasoconstriction increases peripheral resistance, leading to an increase in blood pressure.
4. Angiotensin II stimulates aldosterone release: Angiotensin II also stimulates the release of aldosterone from the adrenal glands. Aldosterone acts on the kidneys, increasing salt and water reabsorption and promoting the excretion of potassium. This leads to an increase in plasma volume and blood pressure.
5. Blood pressure increases: Due to the combined effects of vasoconstriction, increased peripheral resistance, and increased salt and water reabsorption, blood pressure is elevated.
Therefore, the correct statement would be: When renin is released from the juxtaglomerular cells, angiotensinogen is converted into angiotensin I, angiotensin I is converted to angiotensin II, and blood pressure increases.