a 36 year of woman has a disease that affects the levels of acetychlorine in her synapses and neuromuscular junctions. what is a potential consequence of this disease
A. deterioration of the sodium potassium pump
b. abnormal conduction of signals of neurons to muscles
c. inability of neurons to depolarize
d. malfunction of the myelin sheath
Since acetylcholine is the only neurotransmitter used in the motor division of the somatic nervous system, we can expect an abnormal conduction of signals of neurons to muscles.
A potential consequence of a disease that affects the levels of acetylcholine in synapses and neuromuscular junctions is the abnormal conduction of signals from neurons to muscles. To understand this, let's break down the options provided:
A. Deterioration of the sodium-potassium pump: The sodium-potassium pump is responsible for maintaining the proper concentrations of sodium and potassium ions inside and outside the cell. While it is an essential process for nerve function, the given disease does not directly affect this pump, so it is not a potential consequence.
B. Abnormal conduction of signals from neurons to muscles: Acetylcholine is a neurotransmitter responsible for transmitting signals from neurons to muscles at the neuromuscular junction. If the disease affects the levels of acetylcholine, it can disrupt the normal signaling process, leading to abnormal conduction of signals between neurons and muscles. Therefore, option B is a potential consequence.
C. Inability of neurons to depolarize: Neurons depolarize when there is a change in their electrical charge, enabling the transmission of signals. While acetylcholine does play a role in depolarization, the given disease specifically affects the levels of acetylcholine, not the ability of neurons to depolarize. Therefore, this option is not a potential consequence.
D. Malfunction of the myelin sheath: The myelin sheath is a protective covering around nerve fibers responsible for enhancing the transmission speed of electrical signals. The disease described does not directly affect the myelin sheath, so this option is not a potential consequence.
Based on the above analysis, the potential consequence of the disease affecting acetylcholine levels is abnormal conduction of signals from neurons to muscles (option B).